To explore the potential sponge activity of recently annotated lncRNAs in the miRNA mediated gene regulation networks at human genome wide scale. Unlike other ceRNA databases, lnCeDB includes but is not limited to miRNA targets on protein coding and lncRNA transcripts predicted from Ago interaction sites within them. The advantage is that it reduces the false positive target detection in our miRNA-target interaction dataset and enhances the reliability of the prediction. At the same time, the dataset is not limited to predictions from only a few cell lines where AGO PAR-CLIP were performed. Also, in lnCeDB we considered, for the first time, that ceRNA activity Cryptochlorogenic-acid largely depends on the relative concentration of the components of a ceRNA network, i.e. the pair of competing RNAs and also the miRNAs they compete for. The provision for checking the tissue specific expression for a potential ceRNA pairalong with the coexpression of Nitroprusside disodium dihydrate shared miRNAs, gives the user a higher chance of identifying the most likely ceRNA candidates in a tissue of interest. One interesting example of a putative ceRNA pair identified by lnCeDB is the lncRNA maternally expressed 3and a transcriptof the protein coding gene Myeloid Cell Leukemia Sequence 1. MEG3 is a maternally expressed imprinted gene encoding a number of alternatively spliced lncRNA transcripts. It interacts with the tumour suppressor P53, and is supposedly a tumour suppressor itself. MEG3 is expressed in many normal tissues including breast, colon, liver, ovary but its expression is lost in many tumour cells. Interestingly, it has been reported that MEG3 is targeted by miRNAs. MCL1 is a member of the BCL2 family and it has three isoforms. The longer isoform is anti-apoptotic whereas the shorter isoforms are pro-apoptotic. The ceRNA pair MEG3-MCL1 putatively shares 16 common miRNAs including miR-28, miR-181d, miR520a, miR-520b and miR-876-3p and show comparable high coexpressions in breast and colon, especially in colon : MCL1 ). The co-expression pattern of MEG3 and MCL1, along with the co-expressed shared miRNAs indicates that MEG3 may act as a ceRNA to MCL1 in colon. Interestingly, the MEG3 gene locus has been reported to be hypermethylated in colorectal cancer cellsindicating the possible perturbation of MEG3 lncRNA expression in colorectal carcinoma. Furthermore, in a colorectal cancer cell, the anti-apoptotic MCL1 has been reported to be regulatedby a number of miRNAs, including miR876-3pwhich we predicted to be shared by MEG3 and MCL1. Together, these observations suggest that there may be a disruption of the potential MEG3-MCL1 ceRNA network in colorectal cancer cells as opposed to the normal colon cells. This observation, however, needs to be validated by further investigations. This example shows the importance of lnCeDB over other ceRNA databases as no other ceRNA database allows the users to check the co-expression patterns of the competing RNAs and the shared miRNAs in different tissue types. Some other interesting observations from lnCeDB are MEG3 and CMPK1 as potential ceRNA pair with near-equal expression signature in colon and ovary and MALAT1-PRKACB potential ceRNA pair in liver. We believe this database will help researchers in deciphering the larger and more complex scenario of miRNA mediated gene regulatory networks in human in the real world of ceRNAs. Collagen peptidesare the hydrolysate components of collagen and are known to have efficacy against various pathologic conditions.